Studies have shown that a gluten-free diet reduces

the risk of type 1 diabetes. So is gluten really that bad?

The connection between diabetes and intestinal microbiota

Research shows that the catalyst for diabetes in the human body are bacteria that inhabit the digestive system (mainly the large intestine). Bacteria affect the immune

system, which, if stimulated too much, will begin to attack other tissues. This condition may lead, among other things, to damage to the pancreas, which results in

impaired insulin production. This is how diabetes develops in the body. Scientists from Copenhagen have been studying the development of bacterial flora in the

digestive system at an early stage of life for years. They proved, among other things, that the bacteria that inhabit our digestive system are largely inherited from our

mother, who passes them on to us during childbirth. This is also confirmed by studies in which mice born naturally had a different bacterial flora than those born by

cesarean section. It has also been shown that the lack of gluten in the diet allows for the free growth of the so-called good bacteria that are in harmony with our immune

system. Following this lead, scientists decided to check whether health control through diet, which is actually mediated by bacteria, can be inherited. We therefore

investigated whether mice on a gluten-free diet would be in better shape than those fed without any changes, and whether their children would also inherit good

bacteria and thus reduce their chances of developing type 1 diabetes.

The occurrence of diabetes and gluten consumption

The results exceeded all scientists' expectations. Not only did children born to mothers on a gluten-free diet have a reduced chance of developing diabetes, but so did

their children, the next generation of mice taking part in the experiment. The F2 generation showed better immune system parameters, and as a result, their chances of

developing diabetes were also reduced. These studies allow the identification of groups of bacteria associated with an increased risk of various diseases, which in the

future may lead to the development of a plan for their health-promoting regulation, e.g. through diet. Such knowledge would enable the use of an appropriate nutritional

model that would help protect against the occurrence of a given disease. According to research by Danish scientists, a gluten-free diet introduced into the daily menu of

mice ensured the absence of type 1 diabetes, both in parents and in subsequent generations. But on the other hand, an appropriately defined diet, including gluten, but

aimed strictly at strengthening the action of specific groups of positive bacteria previously selected for a given individual, can bring the same effect, i.e. protect him or

her from type 1 diabetes. That is, building a given individual through a proper diet specific microbiota, containing bacteria that are beneficial for the body's immune

system, will not only help avoid diabetes, but will protect it against a number of other illnesses and diseases. This approach seems to be much better than a restrictive

diet, especially in the case of diabetics.


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